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Catalogue of Gene Symbols

Gene Symbol list

Hit Count : 28

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Gene symbol Class Synonyms Chromosomes Germplasms
Level1 Level2 Level3 Level4
QFhs.pur-7El Reaction to Fusarium spp. Disease: Fusarium head scab, scab 7el2,7DS.7DL-7el2 su:K2630,v:K11695 = 7DS.7DL-7el2,v:KS10-2 = 7el2S.7el2L-7DL,v:KS24-1 and KS24-2 = 7DS.7el2,ma:Qfhs.pur-7el2 was flanked by BE445653 and Xcfa2270-7D,ma:These markers were also present in KS10-2
Comment:
 
Fhb2 Reaction to Fusarium spp. Disease: Fusarium head scab, scab 6BS v:pbE85,v2:BW278 Fhb1,v2:Sumai 3 Fhb1,ma:Xgwm133-6B - 4 cM - Fhb2 - 2 cM - Xgwm644-6B
Comment:
The relationship of Fhb2 to Fhs1 or Fhs2 {1096} is unknown.  
QFhs.inra-6D Reaction to Fusarium spp. Disease: Fusarium head scab, scab ma:Associated with Xcfd42-6D (LOD=2.7, R2=6.6%)
Comment:
 
QFhs.ndsu-3B Reaction to Fusarium spp. Disease: Fusarium head scab, scab 3BS v:Sumai 3/Stoa RI mapping population; the QTL was contributed by Sumai 3,ma:Association with Xbcd907-3B.2 (LOD>3) {9925} and microsatellite markers Xgwm1533-3B and Xgwm493-3B,ma:QFhs.ndsu-3B from Sumai 3 was associated with microsatellite loci Xgwm533-3B and Xgwm274-3B in certain Sumai 3 derivatives {10062}. In Ning 894037 the QTL has the same location and similar SSR bands to Sumai 3 {10085}. STS marker SRST.3B1 was mapped between Xgwm533-3B and Xgwm389-3B and associated with QFhs.ndsu-3B {10072}. QFhs.ndsu.3B was associated with markers Xgwm533-3B, Xbard133-3B, Xbarc147-3B and Xgwm493-3B
Comment:

This QTL explained 42% of the variation in Sumai 3/Stoa{0175}.
Two additional QTL for resistance to Fusarium graminearum were identified in the cross Sumai3/Stoa {0175}. The QTL on 4BS was associated with Xwg909-4B and the QTL on 6BS was associated with Xbarc101-6B and Xbcd1383-6B {0175}. The QTL associated with markers Xgwm493-3B/Xgwm533-3B (explaining 24.8 % of the variation), and Xbarc101-6B/Xbcd1383-6B were also identified in a RIL population from the cross ND2603/Butte 86 {0175}. In addition, one QTL on chromosome 3AL associated with Xbcd941-3A and one on chromosome 6AS associated with XksuH4-6A were identified in RILs from the cross ND2603/Butte 86 {0175}.
Resistance QTL on chromosome 3BS associated with Xgwm493-3B and Xgwm533-3B was also identified in a DH population of the cross Remus/CM-82036 (a Sumai 3 derivative) {0240}. Additional QTL in this cross were detected on chromosome 5A, associated with Xgwm293-5A and Xgwm304-5A, and possibly on 1B, associated with Glu-B1 {0240}.
Two major genes with additive effects were reported in crosses between Sumai 3 (resistant) and two susceptible cultivars{0174}. One of the genes was assigned to 5AL based on linkage to the dominant awn suppressor B1 (RF 15.1-21.4%).
QTLs were located in 3BS, 2BL and 2AS in Ning 7840/Clark. The most effective QTL was probably in the interval flanked by deletions 3BS-3 and -8 and was close to Xgwm533-3B and Xbarc147-3B {0328}.
A marker study found that 14 of 66 wheats with putative FHB resistance shared markers indicative of the 3BS QTL in Ning 7840, Sumai 3, Wangshuibai and possibly Wuhan 3, plus Japanese landraces Shinchunaga and Shirasu No 1 {10115}. The original source may be the landrace 'Taiwan Wheat' rather than Funo {10115}. Four QTLs on chromosomes 3BS (associated with Xbarc133-3B), 3BL (Xgwm247-3B) and 3AS (Xgwm5-3A) from Huapei 57-2, and 5BL (Xbarc59-5B) from Patterson, were reported in the cross Huapei 57-2/Patterson {10026}. Huapei 57-2, Ning 7840 and Sumai 3 carried common alleles in the Xgwm533-3B, Xgwm493-3B, Xbarc147-3B and Xbarc133-3B region {10026}.
Wuhan-1/Nyubai{10623}: Two QTLs were located on chromosomes 2DL and 3BS (distal) {10020}.
Field resistance
G16-92(R)/Hussar(S): Two QTL for resistance to F. culmorum were identified on chromosome 1A (resistance from Hussar) (R2 = 0.01) and 2B (resistance from G16-92) (R2 = 0.14) {10588}.
Of 54 lines with reported FHB resistance, 6, including CM-82036, Ning 7840 and Wuhan 3, had the same 5-marker haplotype as Sumai 3, and 4 lines possessed 4 of the markers. Twenty-nine lines, including Frontana, had no marker allele in common with Sumai 3, whereas 13 lines had 1 to 3 alleles in common with it {10113}. Qfhs.ndsu-3B and the 5 marker loci were placed in 3BS deletion bin 0.78-0.87 {10144}.
Nanda2419(S)/Wangshuibai(R): 8 QTLs were identified; those with large effects were associated with Xgwm533-3B.3 - Xgwm533-3B.1 (W), Xwmc539-6B (W) and Xs1021m-2B - Xgwm47-2B {10190}.
Type I resistance (% infected plants) in this cross was attributed to 10 chromosome regions among which Qfhi.nau-4B (Xwmc349-4B - Xgwm149-4B - r2=0.75), XFhi.nau-5A (Xwmc96-5A - Xgwm304-5A - R2=0.27) and Qfhi.nau-5B (Xgwm408-5B - Xbarc140-5B) from Wangshuibai were detected in at least 3 of 4 years {10282}. A significant additive effect of QTL on 6D and 2A was also observed {10282}.
Type IV resistance (proportion of Fusarium-damaged kernels) was attributed to five QTLs, four from Wangshuibai. Those with the largest effects included QFdk.nau-2B (from Nanda 2419), QFdk.nau-3B and QFdk.nau-4B {10577} with each accounting for more than 20% of the phenotype variation.
Pelikan (S)/G93010 (=Bussard / Ning 8026) (R). Qfhs.Ifl-7BS/5BL and Qfhs.Ifl-6BS (probably Fhb2) from Ning 8026 reduced disease severity by 30% and 24%, respectively, and by 46% when combined {10594}. Other resistance genes were located on chromosomes 1AS (Qfhs.Ifl-1AS from Pelikan), and 2AL and 7AL (from Ning 8026) {10594}.
Spark (MR)/Rialto(S) DH population: Of nine QTLs identified across all environments, seven alleles for resistance came from Spark and two from Rialto. The largest effect on Type 1 resistance (Xfhs.jic-4D.2) was associated with the Rht-D1b allele in Rialto which made lines more susceptible. Other QTLs occurred on chromosomes 1B (1B.1R), 4D (Qfhs.jic-4D.2), 2A,3A (each, 2 QTLs), 5A and 7A. Xfhs.jic-4D.2 had little effect on Type 2 resistance {10603}.
Wangshuibai/Seri 82:F3:F5 population: QTL on chromosome 3BS (Xgwm533-3B - Xs18/m12-3B) and 2DL(Xgwm539-2D - Xs15/m24-2D) accounted for 17% and 11%, respectively, of the phenotypic variance {10264}.
Wangshuibai/Alondra 'S': A stable QTL was associated with Xgwm533-3B in each of 3 years, QTLs in 5B (Xgwm335-5B), 2D and 7A were detected in 2 years {10268}.

Wangshuibai(R)/Wheaton(S): QTLs located in chromosome 3BS (Xbarc147-3B, R2=37% & Xbarc344-3B, R2=7%), 7AL (Xwms1083-7A, R2=10%) and 1BL (Xwms759-1B, R2=12%) {10200}.
Chokwang (R)/Clark (S):
Qfhb.ksu-5DL.1 associated with Xbarc239-5D (R2=0.24) {10276}, Qfhb.ksu-4BL.1 associated with Xbarc1096-4B (R2=0.13) {10276}, and Qfhs.ksu-3BS.1 marginally associated with the region of Fhb1 (R2=0.1) {10276}.
Ernie(Res)/MO94-317(Sus): 243 F8 RIL population. Four QTLs from Ernie detected as follows:
Qfhs.umc-2B, linked to Xgwm278-2BS, R2 = 0.04 {10456}.
Qfhs.umc-3B, linked to Xgwm285-3BS, R2 = 0.13 {10456}.
Qfhs.umc-4B, linked to Xgwm495-4BL, R2 = 0.09 {10456}.
Qfhs.umc-5A, linked to Xgwm165-5A, R2 = 0.17 {10456}.
Evidence was provided to suggest the QTL acted additively {10456}
Associations between responce to FHB caused by F. culmorum and the semi-dwarfing locus Rht-D1 in crosses Apache/Biscay, Romanus/Pirat and History/Rubens (Biscay, Pirat and Rubens carry Rht-D1b) were reported in {10574}. Genotypes with the semi-dwarf alleles tended to be more susceptible.
A review of 52 mapping studies is provided in {10593}.
Seedling resistance to Fusarium graminearum (FSB). A QTL for FSB resistance in the Wuhan/Nyubai population was associated with the Qwmc75-5B locus, R2 = 0.138. The relationship of this resistance to crown rot resistance is unknown {10624} (see Reaction to F. pseudograminearum).
Tetraploid wheat
Langdon/Langdon (DIC-2A) RICL population: Increased susceptibility of the T. dicoccoides Israel A substitution line relative to Langdon was mapped to a 22 cM interval spanned by Xgwm558-2A and Xgwm445-2A {10613}.  
Fhb3 Reaction to Fusarium spp. Disease: Fusarium head scab, scab 7DS = T7AL.7Lr#1S v:TA 5608,al:Leymus racemosus,ma:Three PCR markers, Be586744-STS, BE404728-STS and BE586111-STS, were developed
Comment:
The level of type 2 resistance conferred by Fhb3 was similar to that of Sumai 3 {10529}.  
Fhb5 Reaction to Fusarium spp. Disease: Fusarium head scab, scab Qfhi.nau-5A 5AS bin:C-5AS3-0.75,i:Mianyan 99-323 and PH691 backcross derivatives selected for Qfhi.nau-5A,v2:Wangshuibai Fh4b,ma:Mapped to a 0.3 cM interval between Xbarc117/Xbarc358/Xgwm293/Xgwm304-5A and Xgwm415-5A
Comment:
Closely linked in coupling with Qflw.nau-5A for narrow leaf width, but recombination is reported in {11041}.  
QFhs.inra-5D Reaction to Fusarium spp. Disease: Fusarium head scab, scab ma:Associated with Xcfd29-5D (LOD=4.4, R2=7%)
Comment:
 
Qfhs.ndsu-3AS Reaction to Fusarium spp. Disease: Fusarium head scab, scab sutv:LDN-DIC3A,tv:T. dicoccoides,ma:Located in an interval spanning 29.3 cM this QTL accounted for 37% of the phenotypic variation; peak marker, Xgwm2-3A
Comment:
 
QFhs.inra-3B Reaction to Fusarium spp. Disease: Fusarium head scab, scab ma:Associated with Xgwm383b-3B (LOD=5.4, R2=10.5%)
Comment:
 
Qfhs.crc-2BL Reaction to Fusarium spp. Disease: Fusarium head scab, scab tv:Strongfield,ma:Spanning 16 cM, this QTL peaking on Xgwm55-2B explained 23% of the phenotypic variation
Comment:
 
QFhs.inra-5A.2 Reaction to Fusarium spp. Disease: Fusarium head scab, scab ma:Associated with Xgwm639b-5A 8LOD=6.6, R2=14%)
Comment:
 
Qfhs.crc-6BS Reaction to Fusarium spp. Disease: Fusarium head scab, scab tv:T. turgidum var. carthlicum cv. Blackbird,ma:Spanning 23 cM and peaking on Xwmc397 this QTL accounted for 23% of the phenotypic variation
Comment:
 
QFhs.inra-2B Reaction to Fusarium spp. Disease: Fusarium head scab, scab ma:Associated with Xgwm374-2B (LOD=7.6, R2=12%)
Comment:
 
QFhs.inra-2A Reaction to Fusarium spp. Disease: Fusarium head scab, scab ma:Associated with Xgwm382c-2A (LOD=6.3, R2=14.4%)
Comment:
 
Fhs1 Reaction to Fusarium spp. Disease: Fusarium head scab, scab v:Line A,v2:Ning 7840 Fhs2
Comment:
 
Fhb7 Reaction to Fusarium spp. Disease: Fusarium head scab, scab FhbLoP v:SDAU1881,v:SDAU1886,ma:Flanked by 7el2 markers Xcfa2240 and XsdauK66 in a 1.7 cM interval,v:KS24-2
Comment:
Derived from Thinopyrum ponticum  
Qfhs.ifa-5A Reaction to Fusarium spp. Disease: Fusarium head scab, scab 5A v:Remus/CM-82036,ma:Associated with markers Xgwm293-5A, Xgwm304-5A, Xgwm1057-5A, Xbarc117-5A, Xbarc186-5A, Xbarc100-5A and Xbarc40-5A
Comment:
Associated mainly with resistance to fungal penetration {10073}.  
Fhs2 Reaction to Fusarium spp. Disease: Fusarium head scab, scab v:Line B,v2:Ning 7840 Fhs1
Comment:
A major QTL was associated with several linked AFLP markers tentatively located in chromosome 7BL of Ning 7840{0005}.
QTLs for resistance to Fusarium graminearum detected in the cross Renan/Recital {10069}. All resistance alleles, except QFhs.inra-3A, were contributed by Renan. LOD scores and percent of variation explained by the QT (R2) are average of three years of field tests.  
QFhs.inra-5A.1 Reaction to Fusarium spp. Disease: Fusarium head scab, scab ma:Associated with Xpsr170a-5A (LOD=3.8, R2=5%)
Comment:
 
QFhs.inra-5A.3 Reaction to Fusarium spp. Disease: Fusarium head scab, scab ma:Associated with B1 (LOD=6.3, R2=8.5%)
Comment:
 
Qfhs.fcu-7AL Reaction to Fusarium spp. Disease: Fusarium head scab, scab sutv:LDN-DIC 7A,tv:T. turgidum var. dicoccoides PI 78742,ma:Located in an interval 39.6 cM thie QTL accounted for 19% of the phenotypic variation in a RIL population of Langdon/LDN-DIC 7A; nearest marker Xbarc121-7AL
Comment:
Strongfield/T. carthlicum(Blackbird): Field resistance identified in chromosome 2BL (Xgwm55-2B), and 6BL(Xwmc397-6B) (coincident with Fhb2 {10225}.  
Fhb4 Reaction to Fusarium spp. Disease: Fusarium head scab, scab Qfhi.nau-4B 4BL bin:4BL5-0.86-1.00,i:Mianyang 99-323*4/Nanda 2419/Wangshibai,v2:Wangshuibai Fhb5,ma:Located in a 1.7 cM segment flanked by Xhbg226-4B and Xgwm149/Xmag4580-4B
Comment:
Although plants with Fhb-4 were taller than the recurrent parent, the height difference was not associated with the Rht-B1 locus {10885}.  
QFhs.pur-2D Reaction to Fusarium spp. Disease: Fusarium head scab, scab v:Alondra,ma:Located on 2DS between SSR markers Xgwm296-2D and Xgwm261-2D
Comment:
 
Fhb1 Reaction to Fusarium spp. Disease: Fusarium head scab, scab QFhs.ndsu-3BS 3BS i:HC374/3*98B69-147,i:Sumai3*5/Thatcher,v:Alsen,v:HC-147-126,v:Rollag,v2:BW278 Fhb2,v2:Sumai 3 Fhb2,ma:XSTS3B-80 - 0.2 cM - Fhb1 - 1.1 cM - XSTS3B-142,ma:Placed in a 1.2 cM interval flanked by XSTS3B-189 and XSTS3B-206
Comment:
W14(R)/Pioneer 2684(S) population: QTL in 3BS and 5AS accounted for 33%, 35% and 31% of the phenotypic variation for disease spread, kernel infection and DON accumulation in greenhouse experiments, and 34% and 26% of variation for FHB incidence and severity in the field {10239}. Flanking markers were Xbarc133-3B & Xgwm493-3B and Xbarc117-5A & Xbarc56-5A {10239}.
The relationship of Fhb1 to Fhs1 or Fhsb2 {1096} is unknown. Lines combining Fhb1and Sr2 are reported in {11170}; Fhb1 is located about 2 cM proximal to Sr2.

SYN1 / Ocoroni DH population: three QTL from SYN1 were identified, QFhs.cim-2D (PVE 25%), QFhs.cim-7A (PVE 4.7%) and Qfhs.cim-7A (PVE 4.2%) {11165}.  
Fhb6 Reaction to Fusarium spp. Disease: Fusarium head scab, scab 1AS v:TA5660, KS14WRRC61,v:TA5093,ma:Three CAPS and one KASPar SNP (wg1S-snp1) markers were developed,v:TA5655
Comment:
Derived from Elymus tsukushiensis syn. Roegneria kamoji TA5660 is in Chinese Spring background; TA5093 is in Everest background.  
QFhs.ndsu-3AS Reaction to Fusarium spp. Disease: Fusarium head scab, scab 3AS tv:T. turgidum var. dicoccoides. Recombinant substitution lines LDN and LDN(Dic-3A). The resistant allele was contributed by T. dicoccoides,ma:Associated with Xgwm2-3A (explained 37% of the phenotypic variation),ma:QFhs.ndsu-3AS was placed within a 11.5 cM region flanked by TRAP marker loci Xfcp401-3A and Xfcp397.2-3A,ma:This gene is unlikely to be a homoeologue of Qfhs.ndsu-3BS = Fhb1
Comment:
 
QFhs.inra-3A Reaction to Fusarium spp. Disease: Fusarium head scab, scab ma:Associated with Xbcd372-3A (LOD=3.7, R2=6.2%)
Comment:
 
QFhs.ndsu-2A Reaction to Fusarium spp. Disease: Fusarium head scab, scab 2AL v:Sumai 3/Stoa RI mapping population; the QTL was contributed by Stoa,ma:Association with RFLP XksuH16-2A (LOD>3)
Comment:
 
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