‘—§Έ__ŒoƒZƒ“ƒ^[_ŒoŒ€‹†Š@ˆβ“`ŽqHŠwŒ€‹†•”@

National Center of Neurology and Phychiatry,

National Institute of Neuroscience

96 ”N 7 ŒŽ XV

Contact: Matsuzaki Fumio __Όθ@•Ά—Y

Email: f_matsuzaki@ncnpja.ncnp.go.jp

Name


Cytology

Genotype

Function

Origin

abl4

D-abl

abl4, e/ TM6B, Tb, e

kinase, EMS


Hoffmann



abl4,e/ TM3, Sb, e, ryRK

kinase, EMS


Hoffmann

abl/pros


(abl4, prosM4)17/ TM3, Sb, ryRK

double mutation


Hoshi



(abl4, prosM4)18/ TM3, Sb, ryRK

double mutation


Hoshi



(abl4, prosM4) 29/ TM3, Sb, ryRK

double mutation


Hoshi



(abl4, prosC43) 10/ TM3, Sb, ryRK

double mutation


Hoshi



(abl4, prosC43) 16/ TM3, Sb, ryRK

double mutation


Hoshi

amn

amnesiac

19A1 decay

amn[PS801]

quick memory

U600

And

Andante

10E2-E4


abnormal period

Tani

And

Andante

10E2-E4

Df(1)HA85/FM7

abnormal period

U15300

And

Andante

10E2-E4

Df(1)KA6/FM7

abnormal period

U16300

ast

asteroid

21E1-2

ast ho

eyes rough &small

M1431

bib

bigbrain


bib/CyO

neurogenic

Nakao

bas[LM]

bang-sensitive

14B5-C3



Tani

bsk

basket

31B-E

bsk[IIJO4] cn bw sp/CyO


M1499

Cha

Choline-acetyl-transferase

91C

Cha[ts1], Cha[ts2]


Kita

daKN7

daughterless


daKN7/ SM1

proneural

Nakao

Delta2-3



ry506, pp, P[ry+D2-3](99B)

jumpstarter

Hiromi




CyO/sp; ry506, Sb P[ry+D2-3]/TM6, Ubx


Hama

dl

delta


dl/ TM3

neurogenic

Nakao

Ddc

Dopa-decarboxylase

37C1-2

Ddcts2 cn/SM5


I3004

Ddc

Dopa-decarboxylase

37C1-2

Ddcts1/SM1


Tani

den

denervated(da)


den/CyO

proneural

Campos-Ortega

dun

dunce

3D4

dun[1]

defect in olfactory memory

?????




dun[2]


?????

dy

dusky

10E2


dark eye colour

I30

e11

ebony

93D2

e11

b-alanyl-dopamine synthase

Tani




e11


U60400

E(spl)

Enhancer of split

E(spl),e /TM3

neurogenic


Nakao

eag[1]

ether a go-go

12F6-13A4


K+ channel (IK)

I3561

Elp

Ellipse

57C9-D5

Bc Elp/CyO

DER Tyr kinase

M1486

eya

eyes-absent

26E


w/o eyes with ocelli

M1922

F202.1


P[ftz-lacZ, ry](X); ry506

mutator


Hiromi

mbd

mushroom body defect

17A-18A2

Df(1)N19/FM6

defect in olfactory memory

U17000

mnb[1]

mini brain

16E3-F3



Yama

ninaC

neither- nor-after potential C

28A1-3

ninaC[P221]

ser/thr-kinase rhabdomere-microvilli protein

I1867

ninaE


92B6-7

ninaE[P332]

major rhdopsin RH1 of R1-R6

I2103

norpA

no receptor potential A

4B6-C1

norpA[7] (EE5) / FM7A

phospholipase C

Hotta




norpA[12] (SB37)



N55e11

notch


N55e11/FM? bar

neurogenic

Nakao

omb

optomotor-blind

4C5-6

omb/FM6(F) x omb/Y(M)


Yama

pale



pale st e/TM3

octopamine synthase

Tani

para

paralytic

14C7-8

C(1)DX, y f/para[ts]

Na[+] channel

I1326

para

paralytic

14C7-8

C(1)DX, y f/para[ts]

Na[+] channel

U6750

para

paralytic

14C7-8

parats1,parats3, parats115

Na[+] channel

Tani

per-long

period

3B1-2



Tani

per-short

period

3B1-2



Tani

per[01] modifier





Tani

per[01]

period

3B1-2



Tani

per]01]

period

3B1-2



Yama

per[04]

period

3B1-2

per[04]/y f :=


Tani

per[04]

period

3B1-2

per[04]/y f :=


Yama

Ppb[l]



Ppb[l]/SM6a


Tani

prosM4

prospero

86E

prosM4, Ki/TM6B,Tb

neural precursor gene, EMS

Hoffmann

prosA63

prospero

P[ftz-lacZ, ry+](86E)/ TM3, Sb, ryRK

enhancer trap


Koizumi

pros[C1]

prospero

Df (3R) pros[C1]/TM3, Sb, ryRK

ry-revertant of A63 semi-lethal



pros[C7]


Df (3R) pros[C7]/TM3, Sb, ryRK

deletion removing pros structural gene



pros[C18]


Df (3R) pros[C18]/TM3, Sb, ryRK

ry-revertant of A63



pros[C43]


Df (3R) pros[C43]/TM3, Sb, ryRK

ry-revertant of A63 deletion in P[ftzlacZ], no protein



pros[C60]


Df (3R) prosC60/TM3, Sb, ryRK

ry-revertant of A63



psi2

phase-angle2

49E7-50A2

Df(2R)vg[B], vgB/SM5

long period

U46800

psi2

phase-angle2


psi2 b w; st

long period

Yama

rdgA

retinal degeneration A

8A4-C6

rdgA[2] (K014)

diacylglycerol kinase

Hotta

rdgB

retinal degeneration B

12A6-D3

rdgB[8] (KS200)

?

Hotta

rdo

reduced occelli

36E1-3

rdo Ddcl01/CyO

occelli small & colourless often missing

Yama

rdo





M2162

ro

rough

97D3-5

required in signal transduction of R2&R5 toward neighbourig cells.

M2548


rol

reduced optic lobes


rolKS221


Yama

rut

rutabaga

12F5-7

rut [CS]/FM7

adenylate-cyclase

Yama

ry

rosy

87D6-13

ry[506]

xanthine-dehydrogenase

?????




ry[506]


M2602




ry[506]


I 225




ry[506]


U71250

sev

sevenless

10A1-2

sev[LY3]

receptor Tyr kinase w/o R7

Tani

sev

sevenless

10A1-2

w sev

receptor Tyr kinase w/o R7

I1134

Sh[3]

Shaker

16F1-3


K+ channel (IA)

M1198

Sh[5]

Shaker

16F1-3


K+ channel (IA)

I111

Sh[5]

Shaker

16F1-3


K+ channel (IA)

M1199

Sh[6]

Shaker

16F1-3

C(1)DX, y w f/Sh[6]

K+ channel (IA)

U10657

Sh[102]

Shaker

16F1-3


K+ channel (IA)

Tani

Sh[133]

Shaker

16F1-3


K+ channel (IA)

I3563

slit[A32]

slit

52D

P[ftz-lacZ, ry+](52D)/CyO; ry506

midline

our lab

slo

slow poke


slo st

Ca[2+] channel ICF

Tani

so

sine-oculis

43B1-2


w/o ocelli almost no compound eyes

Steller

sol

small optic lobes

19F4

sol[KS58]


Yama

t

tan

8C2-D1


eta-alanyldopamine hydrolase?

Hotta

tipE

temparature-inactivation -induced paralytic E

64A6-64B12

Na[+] channel?

Tani


tur

turnip


tur-87/FM7

GTP-binding-76kDa-protein

Yama

WILD TYPE STRAINS






Canton-S





M3754

Canton-S





I 1

Canton-S





Tani

Canton-S





Okano

Canton-S





Hotta

Oregon-R-C





I 5

Oregon-R





Tani

Oregon-R





Hotta

+ Wild 1A

New York, USA, 1966, B. Wallace




M3647

+ Wild 2A

Ohio, USA, 1966, B. Wallace




M3649

+ Wild 3A

Ohio, USA, 1966, B. Wallace




M3652

+ Wild 5A

Georgia, USA, 2966, B. Wallace




M3654

+ Wild 9A

Georgia, USA, 2966, B. Wallace




M3658

+ Wild 10A

South Carolina, USA, 1966, B. Wallace




M3659

+ Wild 11A

North Carolina, USA, 1966, B. Wallace




M3662

+ Sapporo

7-10-88a, Sapporo, Japan, H. Tanaka




M3665

+5-15-88#a

Nairobi, Kenya, R. C. Woodruff




M3704

ENHANCER TRAP and lacZ MARKER STRAINS






strains


genotypes


lacZ expression

origin

2-1499


2-1499/CyO cn[2] ; ry[506]


pCC

Chiba

2-4560


2-4560/CyO cn[2] ; ry[506]


aCC, not in pCC, RP1,RP3, RP4

Chiba

2-6263


2-6263/CyO cn[2] ; ry[506]


pCC

Chiba

2-10103


2-10103/CyO cn[2] ; ry[506]


pCC

Chiba

F125


F125 on III 95C


Glial precursors

Hiromi

F236


F236 on II ?


Glial precursors

Hiromi

F223


F223 on III 98D


MP2

Hiromi

5743


5743/5743; rf10 / TM3 ry

ftz marker


Hiromi



5743= P[ry+ ; ftz (lacC)] on II




6605


6605/6605; rf10 / TM3 ry

ftz marker


Hiromi



6605= P[ry+ ; ftz (lacB)] on II




6609


6609= P[ry+ ; ftz (lacB)] on X

ftz marker


Hiromi

BALANCERS








CyO/Sco; ry[506]


balancer




CyO/+ ; ry[506]


balancer




w; CxD/ TM3 Sb


balancer

Hama



w; Df (3R)ry[81], e/ TM3, Sb, e, ry[RK]


balancer

our lab.



Df (3R)ry[81], e/ TM3, Sb, e, ry[RK]


balancer

Tani



TM3, Sb, e, ry[RK]/ TM6B, Tb, e


balancer

our lab.

C40, IS3


rf10 / TM2, ry[506], P[lArB]


LacZ-balancer

Gehring



rf10= ftz[9H34], ry [506], e[s]




rf10 / TM3beta


rf10 / TM3 ry[RK], Sb, P[ry+;ftz(lacC)]


LacZ-balancer

Hiromi@

DEFICIENCIES






Df(1)62g18

3A1-2; 3A4-5

Df(1)62g18,y/w[+]Y//C(1)DX, y f/w[+]Y



I1517

Df(1)K95

3A3-4; 3B1-2

Df(1)K95, y2 / C(1)RMy2su(wa)wa ;Dp(1;4)mg / spapol



B3067

Df(1)K95, y[2] cv / C(1)DX,y f / w+Y




U2550


Df(1)64c18

2E1-2; 3C2

Df(1)w64c18, g sd / C(1)DX, y w f / Dp(1;Y)w[+]Y



I936

Df(1)w64c18, g / C(1)DX, y w f / w+ Y




U8500


Df(1)wrj1

3A1-2; 3C2-3

Df(1)w-rJ1/w[+]spl[-]Y//C(1)DX, y w f/w[+]spl[-]Y(not C(1)?)



I1522

Df(1)wrj1, y2 wrj1 ec sn3 / C(1)DX, y f / w+Y




U3450


Df(1)64j4

3A8-9; 3B1-2

Df(1)64J4, y wa spl / C(1)DX, y f / w+Y



U2450

Df(1)64f1

3A9-B1; 3B2-3

Df(1)w64f1, w64f1 / C(1)DX, y f / w+Y



U3350

Df(1)TEM202 / Df(1)64j4

Df(1)TEM202 / Df(1)64J4 / w+Y



M.Young


Df(1)w64d

3B1-2; 3C2-2

Df(1)w64d, w64d / C(1)DX, y f / w+Y



U3345

Df(1)w258-45

3B2-3; 3C2-3

Df(1)w258-45, y w258-45 / FM4



B757

Df(1)w258-45, y w258-45 / C(1)DX, y f / w+Y



U3400



Dp(1;3)wm49a

3B1-2;3E2-3






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