分離済み変異体

tm2905

Allele Nametm2905
BalanceCompleted
OutCrossNot Accepted
Sequence NameF26H9.4
Gene NameF26H9.4
Worm BaseAllele Name tm2905 (x1)
Gene Name F26H9.4
Sequence F26H9.4
Phenotype Information from the receiver is posted in the form of a "researcher : phenotype" lethal or sterile.
Mutation site Please see gene structure to locate the deletion in relation to exon(s) 18699/18700-19219/19220 (520 bp deletion)
ChromosomeI
Putative gene structurecomplement(join(18360..18725, 18932..19055, 19100..19209, 19257..19328))
Map position3.74
BalancerhT2,hT2 [bli-4(e937) let-? qIs48]
Map position of balancer
Sequence of primersIntRev:TCTTGCCGTCGGGTCGTTCA,ExtRev:GTATCCTGATTTTCCACCTG,IntFwd:AACTAACTCCACTGGGACAC,ExtFwd:ATAACCTCATCTGGCGTCGA
Distributed lab
DepositorDr. S. Mitani/NBRP
References Please submit your publication
Kim KW, Tang NH, Piggott CA, Andrusiak MG, Park S, Zhu M, Kurup N, Cherra SJ 3rd, Wu Z, Chisholm AD, Jin Y.
Expanded genetic screening in Caenorhabditis elegans identifies new regulators and an inhibitory role for NAD+ in axon regeneration.
Elife 2018 7  
[ PubMed ID = 30461420 ] [ RRC reference ]

Wang W, McReynolds MR, Goncalves JF, Shu M, Dhondt I, Braeckman BP, Lange SE, Kho K, Detwiler AC, Pacella MJ, Hanna-Rose W.
Comparative Metabolomic Profiling Reveals That Dysregulated Glycolysis Stemming from Lack of Salvage NAD+ Biosynthesis Impairs Reproductive Development in Caenorhabditis elegans.
J Biol Chem 2015 290(43) 26163-79 
[ PubMed ID = 26350462 ] [ RRC reference ]